Innate Immunity: Mechanisms and Modulation

Web Desc
MEETING CHANGE TO VIRTUAL: Innate Immunity: Mechanisms and Modulation
Organizer(s): Kate A. Fitzgerald, Arturo Zychlinsky and Kate Schroder
Date: April 11 - 15, 2021
Location: Virtual at your computer. Recorded presentation content is available On Demand.
Sponsored by AstraZeneca, Cell Research and Genentech, Inc.
Summary of Meeting:
For information on the virtual eSymposia program Click here

The innate immune system is the first line of response to pathogens and tissue injury. Specialized cells have evolved mechanisms to detect microbial and distress signals and translate these into effector mechanisms that fight infections, amplify inflammation, initiate acquired immunity and eventually resolve. Although the innate immune response is usually associated with infectious disease, it has been implicated in a broad range of diseases, including cancer, autoimmunity, degenerative and vascular diseases. This conference provides multidisciplinary perspectives on innate immunity, from fundamental science to clinical aspects of disease, as well as therapeutic approaches to immune modulation. The conference program will focus on recent advances in this rapidly developing field. Presentations will provide new insights into mechanisms of microbial and distress sensing and the effector mechanisms of innate immune cells including macrophages, neutrophils, dendritic cells and innate lymphoid cells. The program will also highlight new approaches to understanding protective and pathogenic innate immune function, and emerging therapeutic opportunities for targeting these mechanisms in disease.
Keystone Symposia thanks our Sponsor(s) for generously supporting this meeting:
AstraZenecaCell ResearchGenentech, Inc.
We appreciate the organizations that provide Keystone Symposia with additional support, such as marketing and advertising:

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Special thanks to the following for their support of Keystone Symposia initiatives to increase participation at this meeting by scientists from underrepresented backgrounds:

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