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Web Desc
Stroke - Molecular, Cellular, Pharmacological and Development of New Therapeutics
Organizer(s): Giora Z. Feuerstein, John M. Hallenbeck and Chung Hsu
Date: March 09 - 14, 2002
Location: Sagebrush Inn & Suites, Taos, NM, USA
Summary of Meeting:
Stroke research has emerged as a discrete discipline that exploits molecular, genetic, cell biology and integrated systems to discover new mechanisms and therapeutics for stroke. In the past few years, novel discoveries in neurogenesis, including neuronal stem cells, provide new insights towards brain plasticity, remodeling and reconstruction. These discoveries provide novel opportunities for brain cell replacement and reconstruction of viable and functional brain tissue. In addition, exciting new research established the presence of inflammatory reaction in brain ischemia. Inflammation bears on diverse aspects of brain response to ischemia including vulnerability, pre-conditioning (tolerance to ischemia) as well as repair, and repair and reconstruction. Such data has already yielded new strategies in design and development of neuroprotective agents, which so far have been based on ion channel modulators and excitotoxic neurotransmitters (e.g., glutamate). Substantial progress has also been made in understanding signaling pathways that govern neuronal survival and death. In particular, understanding the signal transduction pathways of redox molecules, protein kinases (MAPK, src) and caspases (apoptosis) provide fertile grounds for new interventions that enhance survival pathways and/or inhibit death pathways. Finally, the emerging Human (and other animals) genome provides bright prospectives for identification of new molecular targets that could be better exploited for prevention, treatment, diagnosis and prognosis of stroke. The key objective of this meeting is to deliberate on new strategies for stroke therapies based on the innovative research conducted in recent years.
Discounted Abstract Deadline: January 14 2002
Discounted Registration Deadline: January 31 2002
We gratefully acknowledge the generous grant for this conference provided by:

National Institute of Neurological Disorders and Stroke (NINDS)
Grant No. 1 R13 NS44767-01
We appreciate the organizations that provide Keystone Symposia with additional support, such as marketing and advertising:

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Special thanks to the following for their support of Keystone Symposia initiatives to increase participation at this meeting by scientists from underrepresented backgrounds:

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