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Web Desc
Toward Understanding Islet Biology
joint with Obesity: New Insights into Pathogenesis and Treatment
Organizer(s): Lydia Aguilar-Bryan and Markus Stoffel
Date: January 21 - 26, 2003
Location: Keystone Resort, Keystone, CO, USA
Sponsored by Pfizer Global Research and Development
Summary of Meeting:
The pancreatic islet supplies insulin in response to increased blood glucose and is a key player in glucose homeostasis. Immunologic destruction of islets is causative of type I diabetes, while insulin resistance and islet dysfunction are generally ascribed to be the main defects in common type II diabetes. This is the first Keystone symposium focused on islet biology and is designed to highlight and integrate the scientific progress occurring in multiple areas relevant to islet biology, including Beta Cell Differentiation, Growth, Regeneration and Death; Gene Expression in Islets and Transcription Factors; Insulin synthesis, conversion, trafficking and granule biogenesis; Stimulus-Secretion Coupling and Genetics, Autoantigens and Transplantation for type 1 diabetes. The meeting will bring together clinicians and basic researchers from disciplines that traditionally have not had an opportunity to interact with the goal of promoting a cross-fertilization of ideas relevant to islet cell biology. The ultimate objective is to promote bench-to-bedside translation of research to advance management of diabetes.
Discounted Abstract Deadline: September 23 2002
Discounted Registration Deadline: November 21 2002
We gratefully acknowledge the generous grant for this conference provided by:

National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK)
Grant No. 1 R13 DK063897-01
We appreciate the organizations that provide Keystone Symposia with additional support, such as marketing and advertising:

Click here to view more of these organizations
Special thanks to the following for their support of Keystone Symposia initiatives to increase participation at this meeting by scientists from underrepresented backgrounds:

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