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Web Desc
Eicosanoids in Inflammation and Chronic Diseases
Organizer(s): K. Frank Austen, Roy J. Soberman and Jilly F. Evans
Date: January 10 - 15, 2006
Location: Yarrow Resort Hotel, Park City, UT, USA
Supported by The Director's Fund
Summary of Meeting:
The consideration of eicosanoids in chronic inflammatory disease will provide a molecular and structural background that will integrate basic, translational, clinical and therapeutic findings. To focus on eicosanoids as elements in chronic inflammation, it appears instructive to examine three clinical entities in which inflammation is superimposed on pathobiology modulated by genetic and environmental susceptibilities. These entities -- atherosclerosis, rheumatoid arthritis, and bronchial asthma with remodeling -- involve different organ systems, yet share the cellular events of chronicity and evidence for involvement of eicosanoids. Various eicosanoids have been implicated in chronic inflammatory diseases by identification of eicosanoid producing cells within clinical lesions, positional cloning of disease associated candidate genes within the eicosanoid generating pathway, and targeted mutations within the eicosanoid generating pathway that interrupt surrogate animal models of a clinical entity. The identification of two or more receptors for a particular eicosanoid at the cellular membrane of bone marrow-derived inflammatory cells has expanded the role of eicosanoids to all aspects of the innate and adaptive immune host inflammatory responses. The generation of eicosanoids by leukocytes is regulated during cell differentiation and after maturation by local peripheral tissue-derived growth factors and cytokines. Finally, particular eicosanoids can have opposing cellular functions uncovered through the time course of their appearance or the profile of receptor expression on the target cell.
Scholarship Deadline: September 14 2005
Discounted Abstract Deadline: September 14 2005
Abstract Deadline: October 6 2005
Discounted Registration Deadline: November 10 2005
We gratefully acknowledge additional support for this conference from:
Educational donation provided by AmgenNovartis Institutes for BioMedical Research
We gratefully acknowledge additional in-kind support for this conference from those foregoing speaker expense reimbursements:

Educational grant from Millennium Pharmaceuticals, Inc., The Takeda Oncology Company
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Special thanks to the following for their support of Keystone Symposia initiatives to increase participation at this meeting by scientists from underrepresented backgrounds:

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