Alzheimer's Disease: Genes, Cellular Pathways and Therapies joint with Protein Misfolding Diseases: Mechanisms of Misfolding, Pathology and Therapeutic Strategies Organizer(s): Rudolph E. Tanzi and Virginia M.-Y. Lee Date: February 21 - 26, 2006
Location: Beaver Run Resort, Breckenridge, CO, USAAlzheimer's disease (AD), a progressive neurodegenerative disease, is the most common form of dementia in the elderly. Over the past two decades, studies of AD at the genetic, molecular, and cell biological level have revealed a host of genes, proteins, and biological pathways that impact on the pathogenesis of this disease. Characterization of these pathogenic pathways have suggested several promising therapeutic strategies for treating and preventing AD based on curbing the accumulation of the Abeta peptide in brain, regulating tau hyperphosphorylation, modulating inflammatory responses, and enhancing neurotransmitter function. This meeting will cover updates of the genetics of AD, Abeta generation and clearance pathways, regulation of tau, relevant animal models, novel therapeutic strategies, and advances in imaging and biomarkers for AD diagnosis. Scholarship Deadline: October 20 2005 Discounted Abstract Deadline: October 20 2005 Abstract Deadline: November 14 2005 Discounted Registration Deadline: December 21 2005 |