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Web Desc
Diabetes and Metabolic Dysfunction
joint with Mitochondria, Metabolism and Heart Failure
Organizer(s): Jeffrey E. Pessin, Alan R. Saltiel and Deborah M. Muoio
Date: January 27 - February 01, 2015
Location: Santa Fe Community Convention Center, Santa Fe, NM, USA
Sponsored by Intercept Pharmaceuticals, Inc., the Journal of Molecular Cell Biology (JMCB), MedImmune, Novo Nordisk A/S and Pfizer Inc. Supported by an educational donation provided by Amgen.
Summary of Meeting:
Diabetes and obesity-related complications and associated metabolic disorders are major public health problems at the local, national and global levels. These metabolic defects underlie the basis for multiple debilitating diseases including heart disease, stroke, blindness, kidney disease, hypertension, neural degeneration, wound healing, amputations, periodontal disease and birth defects. This meeting will address several cutting-edge aspects of molecular, cellular, tissue and integrative system metabolism that account for the metabolic defects that occur in diabetes and obesity. Several of these themes overlap with the concurrent meeting on Mitochondria, Metabolism and Heart Failure and four concurrent sessions are planned addressing distinct and novel aspects of normal and dysregulation muscle (skeletal and cardiac) intracellular signaling, mitochondria function/dynamics, aging and energy balance. Leaders in each of these respective areas will not only address basic and integrative mechanisms in model systems, but several will address these issues in human pathology. The diabetes-specific sessions reflect several key aspects of metabolic dysregulation in which novel information is currently forthcoming, causing a paradigm shift compared to our previous understanding of these processes. These include new information about tissue cross-talk, the inter-relationship between the control of glucose production and fatty acid synthesis that underlies selective insulin resistance and the role of normal and dysregulated circadian rhythms on metabolic processes. These cutting-edge advances will provide a foundation for identifying future research directions.
Scholarship Deadline: October 1 2014
Discounted Abstract Deadline: October 1 2014
Abstract Deadline: October 28 2014
Discounted Registration Deadline: November 25 2014
Keystone Symposia thanks our Sponsor(s) for generously supporting this meeting:
Educational donation provided by AmgenIntercept Pharmaceuticals, Inc.Journal of Molecular Cell Biology (JMCB)MedImmuneNovo Nordisk A/SPfizer Inc.
We gratefully acknowledge additional support for this conference from:
Journal of Lipid Research
We appreciate the organizations that provide Keystone Symposia with additional support, such as marketing and advertising:
PLOS
We gratefully acknowledge the generous grant for this conference provided by:

National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK)
Grant No. 5R13DK104616-01
We appreciate the organizations that provide Keystone Symposia with additional support, such as marketing and advertising:

Click here to view more of these organizations
Special thanks to the following for their support of Keystone Symposia initiatives to increase participation at this meeting by scientists from underrepresented backgrounds:

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