Autophagy: Molecular and Physiological Mechanisms
Organizer(s): Sharon A. Tooze, Ana Maria Cuervo and Noboru MizushimaDate: June 05 - 09, 2016
Location: Whistler Conference Centre, Whistler, BC, Canada
Autophagy, an evolutionarily conserved pathway, is required for homeostasis, growth and survival in eukaryotic cells. Deregulation of autophagy is implicated in many human diseases, and mutations in autophagy genes are known to be a direct cause of severe human disorders. There are global efforts to understand the role of autophagy in disease pathogenesis and to develop therapeutic strategies to control the autophagic response. However, the latter is hampered by the lack of biomarkers, and disease-specific, selective autophagy targets. Overcoming this barrier in development of therapeutics will require a better understanding of basic autophagy biology. This meeting will specifically address the basic molecular pathway to increase our knowledge of the genes, proteins and signaling pathways involved in autophagy. The program will assemble experts in the molecular details of the diverse autophagic pathways and therefore provide clinicians and pharmaceutical companies with information to develop new, specific targets.
Scholarship Deadline: February 4 2016
Discounted Abstract Deadline: February 4 2016
Abstract Deadline: March 8 2016
Discounted Registration Deadline: April 5 2016
We gratefully acknowledge additional in-kind support for this conference from those foregoing speaker expense reimbursements:
EMBO Young Investigator Programme
Novartis Institutes for BioMedical Research
We gratefully acknowledge the generous grant for this conference provided by:National Cancer Institute (NCI)
Grant No. 1R13CA206357-01
Funding for this conference was made possible (in part) by 1R13CA206357-01 from the National Cancer Institute. The views expressed in written conference materials or publications and by speakers and moderators do not necessarily reflect the official policies of the Department of Health and Human Services; nor does mention of trade names, commercial practices, or organizations imply endorsement by the U.S. Government.