Fairmont Banff Springs Floorplan

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This meeting took place in 2012
Here are the related meetings in 2018:
Mitochondrial Biology (Z1)
For a complete list of the meetings for the upcoming/current season, see our meeting list, or search for a meeting.
Mitochondrial Dynamics and Function (X3)
Organizer(s) Luca Scorrano and Jodi Nunnari
March 19—24, 2012
Fairmont Banff Springs • Banff, Alberta Canada
Abstract Deadline: Nov 17, 2011
Late Abstract Deadline: Dec 21, 2011
Scholarship Deadline: Nov 17, 2011
Early Registration Deadline: Jan 19, 2012
Sponsored by Takeda Pharmaceutical Company Limited
Joint Meeting:
Cell Death Pathways: Beyond Apoptosis (X4)
Summary of Meeting:
Mitochondria are no longer considered “only” as the cellular thermodynamic converter: mounting evidence implicates them as key organelles in cell signaling, proliferation, aging, disease and death. Indeed, our view of this organelle was profoundly altered with the discovery that mitochondria function within a connected network that is continuously remodeled by fusion and division events. This reality has sparked and accelerated new scientific discoveries through the intersection of different fields about the functional roles and molecular features of mitochondrial behaviors. The interaction between the cell death and mitochondrial dynamics fields is the best example of such interplay and has led to the discovery that modulators governing cell death and mitochondrial dynamics, respectively, are in fact interacting with one another. This Keystone Symposia meeting uniquely builds on this rich interdisciplinary interaction, which continues to provide novel insights into the mechanisms underlying mitochondrial behavior and mitochondrial-associated diseases. Opportunities for interdisciplinary interactions will be significantly enhanced by the concurrent meeting on Cell Death Pathways: Beyond Apoptosis, which will share a keynote address and two plenary sessions with this meeting.
View Scholarships/Awards
Mitochondria are no longer considered “only” as the cellular thermodynamic converter: mounting evidence implicates them as key organelles in cell signaling, proliferation, aging, disease and death. Indeed, our view of this organelle was profoundly altered with the discovery that mitochondria function within a connected network that is continuously remodeled by fusion and division events. This reality has sparked and accelerated new scientific discoveries through the intersection of different fields about the functional roles and molecular features of mitochondrial behaviors. The interaction between the cell death and mitochondrial dynamics fields is the best example of such interplay and has led to the discovery that modulators governing cell death and mitochondrial dynamics, respectively, are in fact interacting with one another. This Keystone Symposia meeting uniquely builds on this rich interdisciplinary interaction, which continues to provide novel insights into the mechanisms underlying mitochondrial behavior and mitochondrial-associated diseases. Opportunities for interdisciplinary interactions will be significantly enhanced by the concurrent meeting on Cell Death Pathways: Beyond Apoptosis, which will share a keynote address and two plenary sessions with this meeting.
View Scholarships/Awards
No registration fees are used to fund entertainment or alcohol at this conference
MONDAY, MARCH 19
TUESDAY, MARCH 20
WEDNESDAY, MARCH 21
THURSDAY, MARCH 22
FRIDAY, MARCH 23
SATURDAY, MARCH 24
Conference Program Print | View meeting in 12 hr (am/pm) time
MONDAY, MARCH 19
19:15—21:30
Welcome and Keynote Session (Joint)
Meeting has ended...abstracts no longer viewable online.
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Luca Scorrano,
University of Padova, Italy
08:00—11:15
Mitochondrial Dynamics in Cell Death (Joint)
Meeting has ended...abstracts no longer viewable online.
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Richard J. Youle,
NINDS, National Institutes of Health, USA
Luca Scorrano,
University of Padova, Italy
Integrating Mitochondrial Morphology and Ultrastructure in the Control of Apoptosis: Lessons from Cellular and Mouse Models
Integrating Mitochondrial Morphology and Ultrastructure in the Control of Apoptosis: Lessons from Cellular and Mouse Models
Seamus J. Martin,
Trinity College Dublin, Ireland
Mitochondrial Quality Control and the Bcl-2 Family
Mitochondrial Quality Control and the Bcl-2 Family
Joseph T. Opferman,
St. Jude Children's Research Hospital, USA
Anti-Apoptotic MCL-1 Localizes to the Mitochondrial Matrix and Regulates Mitochondrial Function
Anti-Apoptotic MCL-1 Localizes to the Mitochondrial Matrix and Regulates Mitochondrial Function
Michelangelo Campanella,
RVC and UCL Consortium for Mitochondrial Research, UK
Short Talk: The F1FO-ATPsynthase Inhibitor IF1 Controls Apoptosis by Regulating Mitochondrial Dynamics
Short Talk: The F1FO-ATPsynthase Inhibitor IF1 Controls Apoptosis by Regulating Mitochondrial Dynamics
Nuno Raimundo,
University Medical Center Goettingen, Germany
Short Talk: Mitochondrial Stress Triggers E2F1 Apoptotic Signaling to Cause Deafness – Insights into Tissue-Specific Mechanisms
Short Talk: Mitochondrial Stress Triggers E2F1 Apoptotic Signaling to Cause Deafness – Insights into Tissue-Specific Mechanisms
14:30—16:30
Workshop 1: Non-Apoptotic Death Pathways: Mechanisms and Consequences
*
Carol M. Troy,
Columbia University Medical Center, USA
Patrick A. Lewis,
University of Reading, UK
The ROC Domain of DAPK1 as a Controller of DAPK1 Function and Cell Death
The ROC Domain of DAPK1 as a Controller of DAPK1 Function and Cell Death
Meera Nanjundan,
University of South Florida, USA
Role of SnoN/SkiL in Modulating Cell Death Pathways in the Pathogenesis of Ovarian Cancer
Role of SnoN/SkiL in Modulating Cell Death Pathways in the Pathogenesis of Ovarian Cancer
Sho Morioka,
North Carolina State University, USA
TAK1 Regulates Mitochondrial Integrity, Which Is Important for Preventing TNF-Induced Endothelial Cell Death and Vascular Development
TAK1 Regulates Mitochondrial Integrity, Which Is Important for Preventing TNF-Induced Endothelial Cell Death and Vascular Development
Yuxing Zhao,
St. Vincent's Institute, Australia
Apoptotic and Non-Apoptotic Pathways Lead to Pancreatic beta Cell Destruction in Type 1 Diabetes
Apoptotic and Non-Apoptotic Pathways Lead to Pancreatic beta Cell Destruction in Type 1 Diabetes
Jason Karch,
Cincinnati Children's Hospital Medical Center, USA
Bax and Bak Are Required For Mitochondrial Permeability Transition Pore-Dependent Programmed Cellular Necrosis
Bax and Bak Are Required For Mitochondrial Permeability Transition Pore-Dependent Programmed Cellular Necrosis
17:00—19:00
Building the Organelle: The Import of Protein and Lipids
Meeting has ended...abstracts no longer viewable online.
Jodi Nunnari,
University of California, Davis, USA
The Behavior of Mitochondria
The Behavior of Mitochondria
Hiromi Sesaki,
Johns Hopkins University School of Medicine, USA
Mitochondrial Division Prevents Neurodegeneration
Mitochondrial Division Prevents Neurodegeneration
Thomas Langer,
CECAD Research Center, Germany
Proteolytic Control of Mitochondrial Membrane Dynamics
Proteolytic Control of Mitochondrial Membrane Dynamics
Nils-Goran Larsson,
Max Planck Institute for the Biology of Aging, Germany
Short Talk: Posttranscriptional Regulation of mtDNA Expression in Animals
Short Talk: Posttranscriptional Regulation of mtDNA Expression in Animals
17:00—19:15
Atypical Caspase-Dependent Cell Death Mechanisms
Meeting has ended...abstracts no longer viewable online.
Andreas Villunger,
Medical University Innsbruck, Austria
The PIDDosome in Cell Death and Cell Cycle Regulation
The PIDDosome in Cell Death and Cell Cycle Regulation
David Wallach,
Weizmann Institute of Science, Israel
The ‘Apoptotic’ Caspases as Regulators of Inflammation: New Lessons about Caspase-8 Function
The ‘Apoptotic’ Caspases as Regulators of Inflammation: New Lessons about Caspase-8 Function
Douglas R. Green,
St. Jude Children's Research Hospital, USA
Cutting Both Ways: Why FADD, FLIP, and Caspase-8 are Required for Embryonic Development
Cutting Both Ways: Why FADD, FLIP, and Caspase-8 are Required for Embryonic Development
Kenta Moriwaki,
University of Massachusetts Medical School, USA
Short Talk: Molecular Mechanism and Pathophysiological Roles of RIP3-Dependent Necrosis
Short Talk: Molecular Mechanism and Pathophysiological Roles of RIP3-Dependent Necrosis
Liming Sun,
Institute of Biochemistry and Cell Biology, SIBS, CAS, China
Short Talk: MLKL Mediates Necrosis Signaling Downstream of RIP3
Short Talk: MLKL Mediates Necrosis Signaling Downstream of RIP3
08:00—11:15
Making the Organelle to Work: From Genetics to (Dys)function
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Nils-Goran Larsson,
Max Planck Institute for the Biology of Aging, Germany
Paolo Bernardi,
University of Padova Medical School, Italy
Pathophysiology of the Mitochondrial Permeability Transition
Pathophysiology of the Mitochondrial Permeability Transition
Rosario Rizzuto,
University of Padua, Italy
Identification of the Mitochondrial Calcium Uniporter
Identification of the Mitochondrial Calcium Uniporter
David C. Chan,
California Institute of Technology, USA
Analysis of Mitochondrial Dynamics in Mouse Tissues
Analysis of Mitochondrial Dynamics in Mouse Tissues
Anu Suomalainen Wartiovaara,
University of Helsinki, Finland
Global and Tissue-Specific Consequences of Mitochondrial Dysfunction
Global and Tissue-Specific Consequences of Mitochondrial Dysfunction
Pénélope Andreux,
Amazentis, Switzerland
Short Talk: A Multi-Level Screen in Mammals and the Worm C. elegans for the Identification of Compounds Improving Mitochondrial Function
Short Talk: A Multi-Level Screen in Mammals and the Worm C. elegans for the Identification of Compounds Improving Mitochondrial Function
Vanessa A. Morais,
Katholieke Universiteit Leuven & VIB, Belgium
Short Talk: Parkinson’s Disease: PINK1 Regulates Complex I Activity
Short Talk: Parkinson’s Disease: PINK1 Regulates Complex I Activity
08:00—11:15
Bioinformatics at the Service of the Death-omes
Meeting has ended...abstracts no longer viewable online.
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Maya Saleh,
McGill University, Canada
Andrea Califano,
Columbia University, USA
Interrogating Gene Regulatory Networks to Discover Oncogene and Non-Oncogene Addictions in Cancer
Interrogating Gene Regulatory Networks to Discover Oncogene and Non-Oncogene Addictions in Cancer
Al L. Burlingame,
University of California, San Francisco, USA
Cell Death Pathways: Beyond Apoptosis
Cell Death Pathways: Beyond Apoptosis
Paul J. Muchowski,
University of California, San Francisco, USA
Using Yeast to Identify Pathways that Modify Neurodegeneration
Using Yeast to Identify Pathways that Modify Neurodegeneration
Cole M. Haynes,
University of Massachusetts Medical School, USA
Retrograde Signaling a Mitochondrial Unfolded Protein Response
Retrograde Signaling a Mitochondrial Unfolded Protein Response
Burcin Ikiz,
Trustees of Columbia University in the City of New York, USA
Short Talk: Unraveling the Molecular Mechanism Underlying ALS-Linked Astrocyte Toxicity to Motor Neurons
Short Talk: Unraveling the Molecular Mechanism Underlying ALS-Linked Astrocyte Toxicity to Motor Neurons
Lars Brichta,
Rockefeller University, USA
Short Talk: Comparative Translational Profiling of Dopaminergic Neurons in Mouse Models for Parkinson’s Disease
Short Talk: Comparative Translational Profiling of Dopaminergic Neurons in Mouse Models for Parkinson’s Disease
14:30—16:30
Workshop 1: Extending Mitochondrial Fusion and Fission: Mechanisms and Consequences
Maria Bohnert,
Weizmann Institute of Science, Israel
MINOS, a Novel Mitochondrial Inner Membrane Organizing System
MINOS, a Novel Mitochondrial Inner Membrane Organizing System
Uma Sankar,
University of Louisville, USA
Regulation of the Mitochondrial Fission GTPase Drp1 by the Sulfhydryl Oxidase GFER
Regulation of the Mitochondrial Fission GTPase Drp1 by the Sulfhydryl Oxidase GFER
Axel Niemann,
ETH Zürich, Switzerland
GDAP1-Induced Fission Allows Protection during ROS Stress Conditions
GDAP1-Induced Fission Allows Protection during ROS Stress Conditions
Julio C. B. Ferreira,
Institute of Biomedical Sciences, Brazil
Disruption of Mitochondria Fusion Contributes to Heart Failure; A Role for betaIIPKC
Disruption of Mitochondria Fusion Contributes to Heart Failure; A Role for betaIIPKC
Adam L. Hughes,
University of Utah, USA
A Yeast PINK1/Parkin-Like Mitophagy Pathway
A Yeast PINK1/Parkin-Like Mitophagy Pathway
Jonathon L. Burman,
University of Washington, USA
Selective Vulnerability of Mitochondria in Parkin Mutant Dopaminergic Neurons
Selective Vulnerability of Mitochondria in Parkin Mutant Dopaminergic Neurons
Lindsay DeVorkin,
Deeley Research Centre, Canada
A Drosophila Effector Caspase Mediates Mitochondrial Dynamics and Autophagy
A Drosophila Effector Caspase Mediates Mitochondrial Dynamics and Autophagy
John J. Lemasters,
Medical University of South Carolina, USA
Different Initiators of Mitochondrial Autophagy in Types 1 and 2 Mitophagy
Different Initiators of Mitochondrial Autophagy in Types 1 and 2 Mitophagy
17:00—19:00
The Interface between Mitochondria and the Endoplasmic Reticulum
Meeting has ended...abstracts no longer viewable online.
*
Jodi Nunnari,
University of California, Davis, USA
György Hajnóczky,
Thomas Jefferson University, USA
Mechanisms of ER-Mitochondrial Local Communication
Mechanisms of ER-Mitochondrial Local Communication
Francesco Cecconi,
Danish Cancer Society Research Center, Denmark
ER-Mitochondria Cross-Talk and the Clash between Autophagy and Cell Cycle Progression
ER-Mitochondria Cross-Talk and the Clash between Autophagy and Cell Cycle Progression
Brendan J. Battersby,
University of Helsinki, Finland
Short Talk: Gimap3 Modifying mtDNA Segregation through Autophagy
Short Talk: Gimap3 Modifying mtDNA Segregation through Autophagy
17:00—19:15
Bcl-2 Protein Family
Meeting has ended...abstracts no longer viewable online.
*
Rajiv R. Ratan,
Burke-Cornell Medical Research Institute, USA
Atan J. Gross,
Weizmann Institute of Science, Israel
A Novel Mitochondrial Pathway that Regulates Cell Fate and Function
A Novel Mitochondrial Pathway that Regulates Cell Fate and Function
Ruth M. Kluck,
Walter and Eliza Hall Institute of Medical Research, Australia
How Bak and Bax Permeabilize Mitochondria during Cell Death
How Bak and Bax Permeabilize Mitochondria during Cell Death
Maya Saleh,
McGill University, Canada
The Pro-Apoptotic Protein BID is Required for NOD-Mediated Inflammation and Innate Immunity
The Pro-Apoptotic Protein BID is Required for NOD-Mediated Inflammation and Innate Immunity
Tomomi Kuwana,
La Jolla Institute for Allergy and Immunology, USA
Short Talk: Bax Forms Two Types of Pores in the Mitochondrial Outer Membrane: Slow- and Fast-Release
Short Talk: Bax Forms Two Types of Pores in the Mitochondrial Outer Membrane: Slow- and Fast-Release
Levi J. Beverly,
University of Louisville, USA
Short Talk: Combining Mouse Models, Biochemistry and Drug Screens to Understand the Role BCL2-Proteins in Human Cancer Initiation and Therapeutic Interventions
Short Talk: Combining Mouse Models, Biochemistry and Drug Screens to Understand the Role BCL2-Proteins in Human Cancer Initiation and Therapeutic Interventions
08:00—11:30
Mitochondrial Fusion and Fission: Mechanisms and Regulation
Meeting has ended...abstracts no longer viewable online.
Janet M. Shaw,
University of Utah, USA
Identification of the Minimal Mitochondrial Fission Machinery in Yeast and Humans
Identification of the Minimal Mitochondrial Fission Machinery in Yeast and Humans
Robert A. Screaton,
Sunnybrook Research Institute, Canada
Short Talk: Genome-Wide RNAi Screening Identifies Novel Regulators of Mitochondrial Morphology
Short Talk: Genome-Wide RNAi Screening Identifies Novel Regulators of Mitochondrial Morphology
Antonio Zorzano,
Institute for Research in Biomedicine, Spain
Mfn2 Links Mitochondrial and ER Function with Insulin Signaling
Mfn2 Links Mitochondrial and ER Function with Insulin Signaling
Jennifer A. Lippincott-Schwartz,
Howard Hughes Medical Institute - Janelia Farm, USA
Cell-Cycle Regulation of Mitochondrial Fusion and Fission
Cell-Cycle Regulation of Mitochondrial Fusion and Fission
Xiaodong Wang,
BeiGene, China
Mitochondrial Phosphatase PGAM5 Participates in Necrosis Execution
Mitochondrial Phosphatase PGAM5 Participates in Necrosis Execution
08:00—11:00
Non-Cell Autonomous Mechanisms of Death
Meeting has ended...abstracts no longer viewable online.
Gwenn A. Garden,
University of Washington, USA
Microglia Mediated Neurotoxicity is Downstream of Reactive Oxygen Species, p53 and microRNAs
Microglia Mediated Neurotoxicity is Downstream of Reactive Oxygen Species, p53 and microRNAs
Christian Münch,
Harvard Medical School, The Harper Lab, USA
Prion-Like Propagation of Mutant Superoxide Dismutase-1 Misfolding
Prion-Like Propagation of Mutant Superoxide Dismutase-1 Misfolding
Beata Jablonska,
Children's National Medical Center, USA
FoxO-Dependent Regulation of p27Kip1 Controls Oligodendrocyte Differentiation after Perinatal Hypoxia
FoxO-Dependent Regulation of p27Kip1 Controls Oligodendrocyte Differentiation after Perinatal Hypoxia
Takeshi Watanabe,
University of Tokyo, Japan
Short Talk: Regulation of NR4A Nuclear Receptors by the ASK-p38 MAP Kinase Pathway-Dependent Phosphorylation
Short Talk: Regulation of NR4A Nuclear Receptors by the ASK-p38 MAP Kinase Pathway-Dependent Phosphorylation
14:30—15:30
Workshop 2: Mitochondria on the Move
*
György Hajnóczky,
Thomas Jefferson University, USA
Konrad E. Zinsmaier,
University of Arizona, USA
Miro Controls Mitochondrial Transport, Structure, and Health in Axons through its GTPase or EF-Hand Domains
Miro Controls Mitochondrial Transport, Structure, and Health in Axons through its GTPase or EF-Hand Domains
Richard A. Kahn,
Emory University School of Medicine, USA
Roles of the ARL2 GTPase at Mitochondria
Roles of the ARL2 GTPase at Mitochondria
Ghazaleh Ashrafi,
Harvard University, USA
Damage to Neuronal Mitochondria Arrests their Motility Prior to Mitophagy
Damage to Neuronal Mitochondria Arrests their Motility Prior to Mitophagy
Romain Cartoni,
Children's Hospital Boston, Harvard Medical School, USA
Co-Deletion of PTEN and SOCS3 Enhance Mitochondrial Transport after Axonal Injury
Co-Deletion of PTEN and SOCS3 Enhance Mitochondrial Transport after Axonal Injury
15:30—16:30
Workshop 3: Mitochondria and Endoplasmic Reticulum: Structural and Functional Interplay
Laura L. Lackner,
Northwestern University, USA
An ER-Associated Mitochondria-Cortex Tether Functions in the Distribution and Inheritance of Mitochondria
An ER-Associated Mitochondria-Cortex Tether Functions in the Distribution and Inheritance of Mitochondria
Valentina Debattisti,
Thomas Jefferson University, USA
Marf, the Single Mitofusin of D. Melanogaster, is Selectively Complemented by Mitofusin 2
Marf, the Single Mitofusin of D. Melanogaster, is Selectively Complemented by Mitofusin 2
Heather M. Lamb,
Johns Hopkins Bloomberg School of Public Health, USA
Coexpression of Bax and Bcl-xL in the Secretory Pathway Alters ER Structure
Coexpression of Bax and Bcl-xL in the Secretory Pathway Alters ER Structure
Clement A. Gautier,
Institut du Cerveau et de la moelle Epiniere, France
Novel Role for Parkin in the Regulation of the Physical and Functional Interactions between Mitochondria and the Endoplasmic Reticulum
Novel Role for Parkin in the Regulation of the Physical and Functional Interactions between Mitochondria and the Endoplasmic Reticulum
17:00—19:00
The Interface between Mitochondria, Metabolism and Cell Death
Meeting has ended...abstracts no longer viewable online.
Vamsi K. Mootha,
Massachusetts General Hospital, USA
Discovery of the Mitochondrial Calcium Uniporter via Genomics
Discovery of the Mitochondrial Calcium Uniporter via Genomics
Eileen P. White,
Rutgers University, USA
Mitochondrial Control of Autophagy
Mitochondrial Control of Autophagy
Nika N. Danial,
Dana-Farber Cancer Institute, USA
Programming the Mitochondrial Carbon Substrate Utilization: A Role for BAD
Programming the Mitochondrial Carbon Substrate Utilization: A Role for BAD
Christian Frezza,
Hutchison/MRC Research Centre, UK
Short Talk: Haem Oxygenase is Synthetically Lethal with the Tumor Suppressor Fumarate Hydratase
Short Talk: Haem Oxygenase is Synthetically Lethal with the Tumor Suppressor Fumarate Hydratase
17:00—19:15
Cell Death Therapeutics
Meeting has ended...abstracts no longer viewable online.
*
Douglas R. Green,
St. Jude Children's Research Hospital, USA
Carol M. Troy,
Columbia University Medical Center, USA
Intranasal Cell Permeant Caspase Inhibitors – Therapeutics for Cerebral Ischemia and other Neurologic Disorders
Intranasal Cell Permeant Caspase Inhibitors – Therapeutics for Cerebral Ischemia and other Neurologic Disorders
Loren D. Walensky,
Dana-Farber Cancer Institute, USA
Mining the Repertoire of BH3 Interaction Sites for Next Generation Pro-Apoptotic Therapies
Mining the Repertoire of BH3 Interaction Sites for Next Generation Pro-Apoptotic Therapies
Rajiv R. Ratan,
Burke-Cornell Medical Research Institute, USA
Epigenetic Modifiers Prevent Neurodegeneration: HDACs and Beyond
Epigenetic Modifiers Prevent Neurodegeneration: HDACs and Beyond
Liane Wagner,
University Medical Center Ulm, Germany
Short Talk: Smac Mimetic Sensitizes Glioblastoma Cells to Temozolomide-Induced Apoptosis via Ripoptosome Formation
Short Talk: Smac Mimetic Sensitizes Glioblastoma Cells to Temozolomide-Induced Apoptosis via Ripoptosome Formation
Johannes Noack,
DKFZ - German Cancer Research Center, Germany
Short Talk: Combination of a Sphingosine Kinase Inhibitor and Temozolomide Results in a Strong Synergistic Cytotoxic Effect which Displays Features of Various Cell Death Pathways
Short Talk: Combination of a Sphingosine Kinase Inhibitor and Temozolomide Results in a Strong Synergistic Cytotoxic Effect which Displays Features of Various Cell Death Pathways
08:00—11:15
Mitophagy/Quality Control (Joint)
Meeting has ended...abstracts no longer viewable online.
Mondira Kundu,
St. Jude Children's Hospital, USA
ULK1: Linking Mitophagy with the Cellular Response to Stress
ULK1: Linking Mitophagy with the Cellular Response to Stress
Koji Okamoto,
Osaka University, Japan
Molecular Mechanism of Mitochondria-specific Autophagy in Yeast
Molecular Mechanism of Mitochondria-specific Autophagy in Yeast
Paul A. Ney,
New York Blood Center, USA
A Single Amino in NIX is Essential for Mitochondrial Clearance in Reticulocytes
A Single Amino in NIX is Essential for Mitochondrial Clearance in Reticulocytes
Richard J. Youle,
NINDS, National Institutes of Health, USA
Damage Control - How the Pink1/Parkin pathway can regulate removal of impaired mitochondria by autophagy
Damage Control - How the Pink1/Parkin pathway can regulate removal of impaired mitochondria by autophagy
Rudolf J. Wiesner,
University of Cologne, Germany
Short Talk: Not the Assembly, the Stability of Respiratory Chain Complexes is Impaired by Nonsense Mutations in mtDNA Encoded Respiratory Chain Subunits
Short Talk: Not the Assembly, the Stability of Respiratory Chain Complexes is Impaired by Nonsense Mutations in mtDNA Encoded Respiratory Chain Subunits
Matthew J. LaVoie,
Brigham and Women's Hospital and Harvard Medical School, USA
Short Talk: Parkin Ubiquitinates Bax to Regulate the Apoptotic Response of Mitochondria
Short Talk: Parkin Ubiquitinates Bax to Regulate the Apoptotic Response of Mitochondria
14:30—16:30
Workshop 2: Autophagy in Pathological Conditions
*
John J. Lemasters,
Medical University of South Carolina, USA
Su-Jeong Kim,
University of Southern California, USA
Nuclear Translocation of DJ-1 during Oxidative Stress-Induced Neuronal Cell Death
Nuclear Translocation of DJ-1 during Oxidative Stress-Induced Neuronal Cell Death
Charles A. O'Brien,
University of Arkansas for Medical Sciences, USA
Osteocyte Autophagy Declines with Age in Mice and Suppression of Osteocyte Autophagy Decreases Bone Mass and Mimics Skeletal Aging
Osteocyte Autophagy Declines with Age in Mice and Suppression of Osteocyte Autophagy Decreases Bone Mass and Mimics Skeletal Aging
Jennifer Martinez,
NIEHS, National Institutes of Health, USA
LC3-Associated Phagocytosis is Required for Efficient Clearance of Dead Cells
LC3-Associated Phagocytosis is Required for Efficient Clearance of Dead Cells
Babul Moni Ram,
Centre for DNA Fingerprinting and Diagnostics, India
Calcineurin Inhibitor Cyclosporine A Induces Endoplasmic Reticulum Stress Mediated Non Apoptotic Cell Death Response
Calcineurin Inhibitor Cyclosporine A Induces Endoplasmic Reticulum Stress Mediated Non Apoptotic Cell Death Response
Melanie J. Scott,
University of Pittsburgh, USA
Caspase-1-Dependent Mitophagy Regulates Liver Damage and Cell Death after Hemorrhagic Shock in Mice
Caspase-1-Dependent Mitophagy Regulates Liver Damage and Cell Death after Hemorrhagic Shock in Mice
Melissa Vos,
University of Lübeck, Germany
Vitamin K2 is a Mitochondrial Electron Carrier that Rescues Pink1 Deficiency
Vitamin K2 is a Mitochondrial Electron Carrier that Rescues Pink1 Deficiency
17:00—19:00
Mitochondrial Dynamics and Disease
Meeting has ended...abstracts no longer viewable online.
*
Anu Suomalainen Wartiovaara,
University of Helsinki, Finland
Gerald W. Dorn, II,
Washington University School of Medicine, USA
The Varied Roles of Mitofusins in the Heart
The Varied Roles of Mitofusins in the Heart
Vojo Deretic,
University of New Mexico Health Sciences Center, USA
Autophagy: An Immunity Paradigm with Mitochondria as a Centerpiece
Autophagy: An Immunity Paradigm with Mitochondria as a Centerpiece
Michelle L. Boland,
Medimmune, USA
Short Talk: Bnip3 is Required to Prevent Hepatic Steatosis and Deficiencies in Gluconeogensis through Effects at the Mitochondria
Short Talk: Bnip3 is Required to Prevent Hepatic Steatosis and Deficiencies in Gluconeogensis through Effects at the Mitochondria
17:00—19:00
Use and Interpretation of Assays for Monitoring Mitophagy
Meeting has ended...abstracts no longer viewable online.
*
Paul A. Ney,
New York Blood Center, USA
Orian S. Shirihai,
University of California, Los Angeles, USA
Live Imaging of Mitophagy in Mammalian Cells
Live Imaging of Mitophagy in Mammalian Cells
Hagai Abeliovich,
Hebrew University of Jerusalem, Israel
Mitophagy as a Quality Control Mechanism in Saccharomyces cerevisiae
Mitophagy as a Quality Control Mechanism in Saccharomyces cerevisiae
*Session Chair †Invited, not yet responded.
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