Sagebrush Inn & Suites Floorplan
Registered Attendees
Registered attendees (and speakers, organizers, etc.) will have access to the following items from their Account page:
- Abstracts from speakers and poster sessions, including the joint meeting abstracts, available 30 days prior to the meeting
(You can edit your own abstract from My Account page as well)
NOTE: Abstract authors/submitters may choose to not have their abstract available online and in the secure mobile app until a week before the meeting.
- Full participant list, including joint meeting participants
- Printable Invoices and Invitation Letters
- Scholarship Information
- Lodging Information
Login to My Account page
This meeting took place in 2017
Here are the related meetings in 2018:
Mitochondrial Biology (Z1)
For a complete list of the meetings for the upcoming/current season, see our meeting list, or search for a meeting.
Mitochondria Communication (A4)
Organizer(s) Jared Rutter, Cole M. Haynes and Marcia C. Haigis
January 14—18, 2017
Sagebrush Inn & Suites • Taos, New Mexico USA
Discounted Abstract Deadline: Sep 20, 2016
Abstract Deadline: Oct 13, 2016
Scholarship Deadline: Sep 20, 2016
Discounted Registration Deadline: Nov 14, 2016
Supported by the Directors Fund
Summary of Meeting:
Understanding how mitochondria communicate within the cell will provide important clues to elucidate its roles in normal cell physiology, as well as numerous diseases such as diabetes, neurodegeneration and cancer. The classical view of mitochondria is of an organelle that interacts with other features of cell biology primarily through the provision and consumption of metabolic intermediates and energetic products. Emerging evidence from many areas of science is accumulating to suggest that mitochondria play much more active roles in communication with other organelles, determining cell and organismal behavior. This conference will focus on these integrated behaviors and the resulting communication. The sessions will be organized around the different nodes, modes and destinations of that signaling. It will bring together people and ideas from different areas of cell biology and physiology who typically do not attend the same conferences, resulting in the development of new collaborations and concepts.
View Scholarships/Awards
Understanding how mitochondria communicate within the cell will provide important clues to elucidate its roles in normal cell physiology, as well as numerous diseases such as diabetes, neurodegeneration and cancer. The classical view of mitochondria is of an organelle that interacts with other features of cell biology primarily through the provision and consumption of metabolic intermediates and energetic products. Emerging evidence from many areas of science is accumulating to suggest that mitochondria play much more active roles in communication with other organelles, determining cell and organismal behavior. This conference will focus on these integrated behaviors and the resulting communication. The sessions will be organized around the different nodes, modes and destinations of that signaling. It will bring together people and ideas from different areas of cell biology and physiology who typically do not attend the same conferences, resulting in the development of new collaborations and concepts.
View Scholarships/Awards
No registration fees are used to fund entertainment or alcohol at this conference
The meeting will begin on Saturday, January 14 with registration from 16:00 to 20:00 and a welcome mixer from 18:00 to 20:00. Conference events conclude on Wednesday, January 18 with a closing plenary session from 17:00 to 19:30, followed by a social hour and entertainment. We recommend return travel on Thursday, January 19 in order to fully experience the meeting.
SATURDAY, JANUARY 14
SUNDAY, JANUARY 15
MONDAY, JANUARY 16
TUESDAY, JANUARY 17
WEDNESDAY, JANUARY 18
THURSDAY, JANUARY 19
Conference Program Print | View meeting in 12 hr (am/pm) time
The meeting will begin on Saturday, January 14 with registration from 16:00 to 20:00 and a welcome mixer from 18:00 to 20:00. Conference events conclude on Wednesday, January 18 with a closing plenary session from 17:00 to 19:30, followed by a social hour and entertainment. We recommend return travel on Thursday, January 19 in order to fully experience the meeting.
SATURDAY, JANUARY 14
18:00—20:00
Welcome Mixer
No registration fees are used to fund alcohol served at this function.
08:00—09:00
Welcome and Keynote Address
Meeting has ended...abstracts no longer viewable online.
*
Jared Rutter,
University of Utah, USA
Johan Auwerx,
École Polytechnique Fédérale de Lausanne – EPFL, Switzerland
Cross-Species Genetic Mapping of Targets in Mitochondria, Metabolism and Aging
Cross-Species Genetic Mapping of Targets in Mitochondria, Metabolism and Aging
09:00—11:30
Epigenetic Signaling and Regulation
Meeting has ended...abstracts no longer viewable online.
*
Marcia C. Haigis,
Harvard Medical School, USA
Eyal Gottlieb,
Technion Integrated Cancer Center, Israel
The Onco-Metabolic Role and Liabilities of the TCA Cycle in Cancer
The Onco-Metabolic Role and Liabilities of the TCA Cycle in Cancer
Matthew D. Hirschey,
Duke University, USA
Short Talk: Lipids Reprogram Metabolism to Become a Major Carbon Source for Histone Acetylation
Short Talk: Lipids Reprogram Metabolism to Become a Major Carbon Source for Histone Acetylation
Coffee Break
William G. Kaelin, Jr.,
Dana-Farber Cancer Institute, USA
Molecular Pathogenesis of IDH Mutant Cancers
Molecular Pathogenesis of IDH Mutant Cancers
Atan J. Gross,
Weizmann Institute of Science, Israel
Short Talk: MTCH2: A Critical Regulator of Mitochondria Function and Communication
Short Talk: MTCH2: A Critical Regulator of Mitochondria Function and Communication
14:30—16:30
Workshop 1
*
Valentina Perissi,
Boston University School of Medicine, USA
Adam L. Orr,
Weill Cornell Medical College, USA
Novel Site-Specific Inhibitors of Mitochondrial ROS Production Define ROS-Mediated Events in Health and Disease
Novel Site-Specific Inhibitors of Mitochondrial ROS Production Define ROS-Mediated Events in Health and Disease
Amanda E. Brinker,
University of Kansas Medical Center, USA
Differences in Mitochondrial Haplotype Influence Metastatic Efficiency and Expression of Related Nuclear Genes
Differences in Mitochondrial Haplotype Influence Metastatic Efficiency and Expression of Related Nuclear Genes
Oleh Khalimonchuk,
University of Nebraska, USA
Loss of Mitochondrial Protease OMA1 Alters Proliferative Properties and Promotes Metastatic Growth of Breast Cancer Cells
Loss of Mitochondrial Protease OMA1 Alters Proliferative Properties and Promotes Metastatic Growth of Breast Cancer Cells
Yoshiyuki Tsujihata,
Takeda Pharmaceutical Company, Ltd., Japan
Novel Approach for Exploring Small Molecule Regulators of Mitochondrial ATP under Hypoxia in Cardiomyocytes by Live Cell High Content Screening using ATP Biosensor
Novel Approach for Exploring Small Molecule Regulators of Mitochondrial ATP under Hypoxia in Cardiomyocytes by Live Cell High Content Screening using ATP Biosensor
Allen Kaasik,
University of Tartu, Estonia
Miro Proteins are Required for Priming Mitochondria for PINK1 Induced Parkin Translocation
Miro Proteins are Required for Priming Mitochondria for PINK1 Induced Parkin Translocation
Nicholas R. Weir,
Harvard University, USA
Msp1 Antagonizes Integration of Membrane Proteins into Lipid Bilayers
Msp1 Antagonizes Integration of Membrane Proteins into Lipid Bilayers
17:00—19:15
Mitochondrial/ER Communication
Meeting has ended...abstracts no longer viewable online.
*
Maya B. Schuldiner,
Weizmann Institute of Science, Israel
Jodi Nunnari,
University of California, Davis, USA
ER/Mitochondria Contact Sites as Organizers of Mito Function
ER/Mitochondria Contact Sites as Organizers of Mito Function
Thomas Langer,
CECAD Research Center, Germany
Proteolytic Control of Mitochondrial Function
Proteolytic Control of Mitochondrial Function
Luca Scorrano,
University of Padova, Italy
Unbiased Genetic and Proteomic Screenings Unveil the Mitochondria-Endoplasmic Reticulum Contacts Machinery
Unbiased Genetic and Proteomic Screenings Unveil the Mitochondria-Endoplasmic Reticulum Contacts Machinery
György Hajnóczky,
Thomas Jefferson University, USA
Calcium and ROS Regulation at the ER-Mitochondrial Interface
Calcium and ROS Regulation at the ER-Mitochondrial Interface
19:15—20:15
Social Hour with Lite Bites
No registration fees are used to fund alcohol served at this function.
08:00—11:00
ROS Signaling
Meeting has ended...abstracts no longer viewable online.
*
Adam L. Hughes,
University of Utah, USA
Navdeep S. Chandel,
Northwestern University, USA
Mitochondria as Signaling Organelles
Mitochondria as Signaling Organelles
Gerald S. Shadel,
Yale School of Medicine, USA
Adaptive Responses to Mitochondrial Stress
Adaptive Responses to Mitochondrial Stress
Heide Christine Patterson,
Whitehead Institute, USA
Short Talk: Mitochondrial ROS-Induced Syk Signaling Mediates Brown Adipocyte Development and Thermogenesis
Short Talk: Mitochondrial ROS-Induced Syk Signaling Mediates Brown Adipocyte Development and Thermogenesis
Coffee Break
Heinrich Jasper,
Buck Institute for Research on Aging, USA
Mitochondrial Stress Signaling, Stem Cells and Lifespan Extension: Lessons from Drosophila
Mitochondrial Stress Signaling, Stem Cells and Lifespan Extension: Lessons from Drosophila
Danica Chen,
University of California, Berkeley, USA
Mitochondrial UPR-Mediated Metabolic Checkpoint, Stem Cell Aging and Rejuvenation
Mitochondrial UPR-Mediated Metabolic Checkpoint, Stem Cell Aging and Rejuvenation
11:00—12:00
NIH Funding Opportunities for Mitochondrial Research: Conversation with an NCI Program Officer
Michael Graham Espey,
NCI, National Institutes of Health, USA
17:00—19:00
Mitochondria/Lysosome Communication
Meeting has ended...abstracts no longer viewable online.
*
Oleh Khalimonchuk,
University of Nebraska, USA
David M. Sabatini,
Whitehead Institute for Biomedical Research, USA
Amino Acid Sensing and Mitochondrial Function
Amino Acid Sensing and Mitochondrial Function
Maya B. Schuldiner,
Weizmann Institute of Science, Israel
Keeping in Touch – Discovering and Characterizing New Contact Sites between Organelles
Keeping in Touch – Discovering and Characterizing New Contact Sites between Organelles
19:00—20:00
Social Hour with Lite Bites
No registration fees are used to fund alcohol served at this function.
08:00—11:00
Communicating Mitochondrial Dysfunction
Meeting has ended...abstracts no longer viewable online.
*
Heidi M. McBride,
McGill University, Canada
Cole M. Haynes,
University of Massachusetts Medical School, USA
Preferential Propagation of Toxic mtDNAs by the UPRmt
Preferential Propagation of Toxic mtDNAs by the UPRmt
Marcia C. Haigis,
Harvard Medical School, USA
Mitochondrial Sirtuin Networks
Mitochondrial Sirtuin Networks
Sebastien Herzig,
The Salk Institute for Biological Studies, USA
Short Talk: Regulation of Mitochondrial Dynamics by AMPK
Short Talk: Regulation of Mitochondrial Dynamics by AMPK
Coffee Break
Matt Kaeberlein,
University of Washington, USA
Mechanisms Linking Severe Mitochondrial Disease and Normative Aging
Mechanisms Linking Severe Mitochondrial Disease and Normative Aging
Jared Rutter,
University of Utah, USA
Stress Responsive Mitochondrial Protein Degradation
Stress Responsive Mitochondrial Protein Degradation
17:00—19:00
Mitochondria and Inter-Cellular Communication
Meeting has ended...abstracts no longer viewable online.
*
Cole M. Haynes,
University of Massachusetts Medical School, USA
William B. Mair,
Harvard School of Public Health, USA
Mitochondrial Plasticity Is Required for AMPK-Mediated Longevity
Mitochondrial Plasticity Is Required for AMPK-Mediated Longevity
Heidi M. McBride,
McGill University, Canada
The Cell Biology of Metabolic Flux
The Cell Biology of Metabolic Flux
Meng C. Wang,
Baylor College of Medicine, USA
Short Talk: Microbes Tune Mitochondrial Dynamics to Regulate Host Metabolic Adaptation to Environmental Variations
Short Talk: Microbes Tune Mitochondrial Dynamics to Regulate Host Metabolic Adaptation to Environmental Variations
19:00—20:00
Social Hour with Lite Bites
No registration fees are used to fund alcohol served at this function.
08:00—11:00
Mitochondria and Fuel Catabolism
Meeting has ended...abstracts no longer viewable online.
Sheng Tony Hui,
Princeton University, USA
Exchange of Mitochondrial Substrates Between Tissues
Exchange of Mitochondrial Substrates Between Tissues
Ralph J. DeBerardinis,
University of Texas Southwestern Medical Center, USA
Mitochondrial Metabolism and its Importance in Health and Disease
Mitochondrial Metabolism and its Importance in Health and Disease
Désirée Schatton,
University of Cologne, Germany
Short Talk: The RNA-Binding Protein CLUH is a Master Regulator of Mitochondrial Catabolic Programme Activated upon Nutrient Deprivation
Short Talk: The RNA-Binding Protein CLUH is a Master Regulator of Mitochondrial Catabolic Programme Activated upon Nutrient Deprivation
Coffee Break
*
Dave Pagliarini,
Morgridge Institute for Research at University of Wisconsin-Madison, USA
Short Talk: Diverse Mitochondrial Protein Functions Revealed by Multi-Omic Mass Spectrometry Profiling
Short Talk: Diverse Mitochondrial Protein Functions Revealed by Multi-Omic Mass Spectrometry Profiling
Benjamin Tu,
University of Texas Southwestern Medical Center, USA
Adaptive Catabolism during Cell Proliferation
Adaptive Catabolism during Cell Proliferation
14:30—16:30
Workshop 2
*
Danica Chen,
University of California, Berkeley, USA
Yi Zhang,
NHLBI, National Institutes of Health, USA
Selective Protein Synthesis on the Mitochondrial Surface Drives the mtDNA Selection
Selective Protein Synthesis on the Mitochondrial Surface Drives the mtDNA Selection
Anna M. Schulz,
Memorial Sloan Kettering Cancer Center, USA
Gcn2-Mediated Mitochondria Protection during Metabolic Stress
Gcn2-Mediated Mitochondria Protection during Metabolic Stress
Jonathan Van Vranken,
University of Utah School of Medicine, USA
FASII Activates Oxidative Metabolism in Mitochondria via ACP Acylation
FASII Activates Oxidative Metabolism in Mitochondria via ACP Acylation
Jessica B. Spinelli,
Harvard University, USA
Evaluating the Effect of Ammonium on Cancer Cell Homeostasis
Evaluating the Effect of Ammonium on Cancer Cell Homeostasis
Laurent Le Cam,
Institut de Recherche en Cancérologie de Montpellier, France
The MDM2 Oncoprotein Controls Complex I Activity and Mitochondrial Dynamics Independently of p53
The MDM2 Oncoprotein Controls Complex I Activity and Mitochondrial Dynamics Independently of p53
Martin Ott,
Stockholm University, Sweden
Modulation of Cellular Stress Signaling and Aging by Changes in Mitochondrial Translation Accuracy
Modulation of Cellular Stress Signaling and Aging by Changes in Mitochondrial Translation Accuracy
Veena Prahlad,
University of Iowa, USA
Mitochondria-Regulated Immune Pathway in C. elegans is Neuroprotective
Mitochondria-Regulated Immune Pathway in C. elegans is Neuroprotective
17:00—19:15
Coordination of Mitochondrial Mass and Physiology
Meeting has ended...abstracts no longer viewable online.
*
Kathryn E. Wellen,
University of Pennsylvania, USA
Maulik Patel,
Vanderbilt University, USA
Short Talk: Homeostatic Stress Responses Regulate Selfish Mitochondrial Genome Dynamics
Short Talk: Homeostatic Stress Responses Regulate Selfish Mitochondrial Genome Dynamics
Richard J. Youle,
NINDS, National Institutes of Health, USA
Mitophagy Regulation
Mitophagy Regulation
Valentina Perissi,
Boston University School of Medicine, USA
Short Talk: Regulation of Mitochondrial Biogenesis through Mitochondria Retrograde Signaling and Chromatin Remodeling of Nuclear-Encoded Mitochondrial Genes
Short Talk: Regulation of Mitochondrial Biogenesis through Mitochondria Retrograde Signaling and Chromatin Remodeling of Nuclear-Encoded Mitochondrial Genes
Nektarios N. Tavernarakis,
Foundation for Research and Technology-Hellas, Greece
Mitochondrial Turnover and Homeostasis during Aging
Mitochondrial Turnover and Homeostasis during Aging
19:15—19:30
Meeting Wrap-Up: Outcomes and Future Directions (Organizers)
Meeting has ended...abstracts no longer viewable online.
19:30—20:30
Social Hour with Lite Bites
No registration fees are used to fund alcohol served at this function.
20:00—23:00
Entertainment
Entertainment is not subsidized by conference registration fees nor any U.S. federal government grants. Funding for this expense is provided by other revenue sources.
*Session Chair †Invited, not yet responded.
We gratefully acknowledge support for this conference from:
We gratefully acknowledge the generous grant for this conference provided by:
We gratefully acknowledge additional support for this conference from:
|
|
We appreciate the organizations that provide Keystone Symposia with additional support, such as marketing and advertising:
Click here to view more of these organizations
Special thanks to the following for their support of Keystone Symposia initiatives to increase participation at this meeting by scientists from underrepresented backgrounds:
Click here to view more of these organizations
|
If your organization is interested in joining these entities in support of Keystone
Symposia, please contact: Sarah Lavicka,
Director of Development, Email: sarahl@keystonesymposia.org, Phone:+1 970-262-2690 Click here for more information on Industry Support and Recognition Opportunities. If you are interested in becoming an advertising/marketing in-kind partner, please contact: Yvonne Psaila, Director, Marketing and Communications, Email: yvonnep@keystonesymposia.org, Phone:+1 970-262-2676 |
